. Headache may be a leading symptom of vascular lesions of the brain. Disturbance of cerebral circulation Carisoprodol Mechanism Of Action, as a rule, develops on the Carisoprodol Price background of vascular diseases, mainly atherosclerosis, and high blood pressure. Stubbornness, refusal to revise outdated stereotypes Carisoprodol Mechanism Of Action – these are possible causes of its occurrence.
Cholesterol and other fats in combination with proteins – lipoproteins, or a fat-protein complex circulate Is Carisoprodol Soma in the blood. Qualitatively changing, they Carisoprodol Mechanism Of Action are perceived by the body as autoantigens, against which antibodies and biologically active Carisoprodol Half Life substances are produced (histamine, serotonin), which affects the vascular wall, increasing its permeability, disrupting metabolic processes.
Atherosclerosis is a chronic disease of the arteries of large and medium caliber (elastic and muscular-elastic type), characterized by the deposition and Aspirin / Carisoprodol accumulation in the intim of plasma atherogenic apoprotein-beta-containing lipoproteins with subsequent reactive Carisoprodol Mechanism Of Action growth of connective tissue and the formation of fibrous plaques, manifested by headaches, dizziness. impairment of general well-being.
Clinical forms of atherosclerosis
. In addition to atherosclerosis of the cerebral arteries, the following forms are distinguished Carisoprodol Tablets (according to the process localization):
1) atherosclerosis of the thoracic and abdominal aorta;
2) atherosclerosis of the coronary arteries;
3) atherosclerosis of the renal arteries;
4) atherosclerosis of the mesenteric arteries Carisoprodol Cost;
5) pulmonary atherosclerosis.
. According to the clinical characteristics, there are 2 periods with the corresponding stages.
Period I (preclinical):
1) the stage of vasomotor disturbances;
2) a complex of biochemical disorders.
II period (period of typical clinical manifestations):
1) ischemic stage Carisoprodol Recreational Use;
2) trombonekrotichesky stage;
3) sclerotic stage.
The phases of the course of atherosclerosis are divided into:
1) the progression phase;
2) stabilization phase;
3) the regression phase.
. Atherosclerosis begins with impaired endothelial permeability and migration to the intima of smooth muscle cells and macrophages. These cells intensively accumulate lipids and turn into “unclean cells.” Overloading unclean cells with cholesterol and its Carisoprodol Overdose esters leads to the breakdown of cells and to the release of lipids Carisoprodol 500Mg and lysosomal enzymes into the extracellular space, which leads to the development of a fibrosing reaction. Fibrous tissue surrounds the lipid mass, fibrous-atheromatous plaque is formed. The development of these processes occurs under the influence of two groups of pathogenetic factors Carisoprodol Mechanism Of Action.
Factors contributing to the development of atherogenic hyperlipoproteinemia:
1) risk factors (age Is Carisoprodol A Controlled Substance over 45 years, male gender, smoking, susceptibility to stress, hypertension, diabetes, overweight, physical inactivity, burdened with atherosclerosis, heredity, gout, soft drinking water, etc.);